|
1
|
- Dr. Kathleen Ethridge
- Northeast Texas Community College
|
|
2
|
|
|
3
|
- In asthma, the airways overreact to a stimulus which causes
bronchospasm, edematous swelling of mucous membranes, and copious
production of thick mucus.
|
|
4
|
- Measures peak expiratory flow volume.
- Normal peak flow for adults range from 300 to 700 L/min
- Baseline values are needed for comparison
|
|
5
|
- For client with asthma, notify the physician for symptoms after
treatment:
- Anxiety
- Increased effort of respirations
- Continuous cough
- Respiratory distress
- nasal flaring
- accessory muscles
- pursed lip breathing
- cyanosis
|
|
6
|
|
|
7
|
- Chronic Bronchitis
- Emphysema
|
|
8
|
- Chronic Bronchitis
- Age 40 to 50 year
- Stocky build with no history of weight loss
- Barrel chest
- cyanotic
- Emphysema
- Age 50 to 75
- Cachectic appearance
- Tachypnea
- pink skin color
|
|
9
|
- Chronic Bronchitis
- increased secretions
- edema
- bronchospasm
- thickened bronchial walls
|
|
10
|
|
|
11
|
- Prevention of infections
- Bronchodilators
- Low flow oxygen
- Corticosteroids
- Balance of activities
- Teach self-care
|
|
12
|
- Acute inflammation of the mucous membranes with raw burning pain in
anterior chest region
- Caused by
- inhalation of noxious gases or smoke
- overvigorous suctioning
- harsh coughing
- observe for cough-related syncope
- Treatment is focused on the cause
|
|
13
|
- Acute epiglottitis
- Acute laryngotracheobronchitis (LTB
- Acute spasmodic laryngitis
- Note:
- Do not examine child’s throat, as it may cause airway spasm
(especially in epiglottitis)
|
|
14
|
- Obstruction of a pulmonary artery
- caused by air, fat, or emboli
- Treatment
- bed rest
- oxygen, ventilator
- anticoagulants
|
|
15
|
- Mean Pulmonary Artery Pressure > 25
- Poor Prognosis
- Symptoms
- dyspnea
- fatigue
- chest pain
|
|
16
|
|
|
17
|
- Definition
- Causes
- Develops when interference of lungs expanding
- pleural effusion; tumor, pneumothorax
- chest wall disorders
- airway obstruction
- insufficient pulmonary surfactant
- increased elastic recoil
|
|
18
|
- Viral infection of respiratory tract
- Spread by droplet
- Sudden onset
- Causes fever, muscle aches and cough
|
|
19
|
- Lobar pneumonia
- consolidation in one lobe of one lung
- Lobular or bronchopneumonia
- patchy consolidation throughtout lobes of one or both lungs
|
|
20
|
- Treatment
- outpatient or inpatient
- obtain culture speciments
- appropriate antibiotics
|
|
21
|
- Pneumococcal pneumonia
- sudden onset, chill, fever, chest pain, cough
- Staphylococcal pneumonia
- sudden onset, fever, chills, pain, cough
- Influenzal pneumonia
- Gram-negative
- sudden onset, high fever, chills, pain, dyspnea
|
|
22
|
- Anaerobic bacterial pneumonia
- low-grade fever, dyspnea, crackles, cyanosis
- Legionnaires’ disease
- fever, headache 48 hrs; then high fever, dyspnea
- Mycoplasma pneumonia
- slowly rising fever, headache, cough
- Viral pneumonia
- headache, myalgia followed by high fever, dyspnea, cough
|
|
23
|
- Fungal pneumonia
- usually asymptomatic
- resembles influenza
- Parasitic pneumonia
- immunocompromised client
- cough, dyspnea, chest pain, fever, crackles, night sweats
|
|
24
|
- Pus within the lung tissue
- Bad odor
- Sputum will have a foul taste
|
|
25
|
- Causes and Prevention
- Pathophysiology
- Treatment
|
|
26
|
- Basic treatment
- 2 months of daily doses of isoniazid and rifampin
- plus 1 or 2 additional drugs
- followed with 4 months of isoniazid and refampin
- at least 2 medications are added to a failing TB treatment program
|
|
27
|
- First-Line Drugs
- Isoniazid (INH)
- Rifampin
- Rifapentine
|
|
28
|
|
|
29
|
- Early identification
- Promptly initiate multidrug therapy
- Isolation
- Particulate respirators
|
|
30
|
- Routine TB skin testing
- Surveillance of cases
- Therapeutic regiments based on clinical history and drug-resistance data
|
|
31
|
- 0-4 mm induration is not significant
- 5mm or greater may be
- 10mm or greater is usually considered significant
|
|
32
|
- Coccidioidomycosis
- Histoplasmosis
|
|
33
|
- Definition
- dysfunction precipitated by an obstruction of the exocrine gland ducts,
causing thick mucous secretions
|
|
34
|
- Group of inflammatory lung diseases
- The alveolar wall becomes thick and fibrotic
|
|
35
|
- Characterized by the formation of widespread granulomatous lesions
- Cause is unknown
|
|
36
|
|